Psychoneurological Complications In Children With Facial Nerve Neuropathy

Abstract

The prevalence of facial nerve neuropathy (NLN), according to the WHO, per 1000 population is about 20 cases, and is one of the leading pathologies of the peripheral system. Most of those with literary sources describe clinical cases of the disease, relying on the forms of lesion, primary and secondary. Primary, or idiopathic NLN occurs up to 90%, respectively, according to the etiological factor in percentage in the first place. The cause of the disease, according to modern scientific research, involves a connection with viruses, the herpes virus occupies a leading position. As a result of the reactivation of the virus, the immune system begins to produce antibodies, as a result, causing an inflammatory reaction in the narrow fallopian canal and, as a result, damage to the facial nerve. During this period, there is an acute demyelization of the nerve trunk. Along with the viral theory, there are theories of the development of ILN, such as hereditary, vascular, immune, compression, lymphatic, the theory of histocompatibility of antigens. The pathological mechanism of NLN, therefore, is complex-ischemic, with dysfunction of the vascular apparatus in the areas of the bone canal and autoimmune, leading to demyelization.